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CAPSAITHIN®-Yongyi
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INCORPORATE IN NORTH AMERICA

Registration address

1260 E Locust St. Suite 302 Ontario, California 91761,United States

Operation Address

Local Business 702-350-7698
Email info@genabolix.com
sales@genabolix.com
Service Email service@genabolix.com
Customer Service customer@genabolix.com
510-431-9888

SHANGHAI OFFICE

Address

Room 1604,Block B,NewCaohejing International Business Center, No.391 Gui Ping Road,Shanghai 200233,China

Office Phone 0086-21-60904639
Email info@genabolix.com
sales@genabolix.com
Customer Service customer@genabolix.com
510-431-9888

Genabolix Canada office

Address

1405-5288 Melbourne Street, Vancouver V5R 6E6, Canada

Email: sales@genabolix.com
Customer Service: customer@genabolix.com
510-431-9888

Genabolix Europe office

Address

Grensstraat 4, 4645 BS Putte, The Netherlands (Nederland)

Attn: Jan de Heij
Tel: 00 31 (0)164603336
Email: jan@genabolix.com
Customer Service: customer@genabolix.com
510-431-9888

Genabolix South Africa office

Address

Rivonia Boulevard, Rivonia Road, Sandton, Johannesburg South Africa

Attn: Raj Bachulal
Tel: +27824805900
Email: raj@genabolix.com
Customer Service: customer@genabolix.com
510-431-9888

CAPSAITHIN®

The effects of dietary capsanthin, the main carotenoid in paprika (Capsicum annuum), on lipid metabolism were examined. Young male Wistar rats were fed diets containing paprika powder, paprika organic solvent extract, residue of paprika extract, and purified capsanthin. Administration of purified capsanthin for 2 weeks resulted in a significant increase in plasma HDL-cholesterol (P < 0·05) without detectable differences in plasma total cholesterol and TAG concentrations. A statistically significant correlation (r 0·567; P < 0·001) was found between dietary capsanthin concentrations and plasma HDL-cholesterol concentrations. Animals receiving diets containing two different capsanthin concentrations exhibited dose-dependent increases in plasma HDL-cholesterol (r 0·597; P < 0·005). While capsanthin was absent in the liver of animals fed the basal diet, it increased markedly in capsanthin-fed animals (P < 0·001). Quantitative analyses of hepatic mRNA levels revealed that capsanthin administration resulted in up-regulation of mRNA for apoA5 and lecithin cholesterol acyltransferase (LCAT), without significant differences in other mRNA levels related to HDL-cholesterol metabolism. These results suggest that capsanthin had an HDL-cholesterol-raising effect on plasma, and the potential to increase cholesterol efflux to HDL particles by increasing apoA5 levels and/or enhancement of LCAT activity.

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